How To Tell If You Have Gastritis. Gastritis is an inflammation of the stomach lining or mucosa. The inflammation may be caused by viral infection, alcohol, smoking, certain drugs, poisoned food, or stress. There are many forms of gastritis. Erosive gastritis can lead to significant bleeding of the stomach and ulcerations. Gastritis may be acute or chronic. It can occur with any major physical stress such as severe illness, surgery or burns, and therefore is referred to as stress- induced gastritis. Medications that are well- known stomach irritants can contribute to gastritis. These include aspirin, nonsteroidal agents such as ibuprofen and pancreatic enzymes. Excessive alcohol ingestion can also cause gastritis and erosions. Sites like alcohol- rehab- nj. ADVERTISEMENTSAcute gastritis causes vomiting, hairy tongue, thirst, severe stomach pain, and mild fever. Dehydration may occur. Definition and facts about gastritis; What is gastritis? What are the signs and symptoms of gastritis? What causes gastritis? Foods to avoid with gastritis (gastritis. The term gastritis means that the lining of the stomach is inflamed. It is an inconvenient and uncomfortable condition, but it is quite treatable. Gastritis is inflammation of the lining of the stomach. It may occur as a short episode or may be of a long duration. There may be no symptoms but, when symptoms are. Chronic gastritis usually produces few symptoms, although in some cases a person may experience one or more of the following discomforts: mild indigestion; slight nausea; a bloated feeling after a small meal; a bad taste in the mouth; and vague stomach pain. Acute gastritis improves of its own accord if the precipitating factor or factors are eliminated. Antacid preparations and/or histamine blockers and antinauseant drugs such as Mylanta or Maalox, and Tylenol; don't use Aspirin, are often prescribed. Don't eat solid foods on the first day of the attack, give your stomach a rest and drink liquids only, milk or water are preferred. Gastritis is a group of various conditions that have one thing in common; inflammation in the stomach lining. Find out the symptoms, causes, remedies and diet for. Do you suffer from gastritis and the terrible pain that comes along with it? Have you considered changing your food habits and menu to get relief from this pain? Add bland foods to your diet slowly and as tolerated (cooked cereals, bananas, rice, potatoes, toast) and avoid greasy, spicy foods. Chronic gastritis can be treated only by eliminating the causative factor, for example, alcohol, smoking, or highly spiced or other foods that are difficult to digest. Antacid drugs are recommended for the treatment of chronic gastritis. The classification of gastritis can depend on the region or function, predominantly involved. Gastric atrophy is a condition which is typically characterised by loss of function, namely reduced acid and intrinsic factor secretion. The combination of changes in the mucosa and the greater risk of bacterial colonisation in the stomach from the low acidity both give rise to gastritis, often with Helicobacter colonisation and an increased risk of carcinoma. Lack of production of intrinsic factor can give rise to pernicious anaemia from decreased Vitamin B1. Gastritis - Wikipedia. Gastritis. Micrograph showing gastritis. H& E stain. Specialty. Gastroenterology. Symptoms. Upper abdominal pain, nausea, vomiting, bloating, loss of appetite, heartburn. It may occur as a short episode or may be of a long duration. There may be no symptoms but, when symptoms are present, the most common is upper abdominal pain. Less common causes include alcohol, smoking, cocaine, severe illness, autoimmune problems, radiation therapy and Crohn's disease, among others. Other conditions with similar symptoms include inflammation of the pancreas, gallbladder problems, and peptic ulcer disease. However, upper centralabdominal pain is the most common symptom; the pain may be dull, vague, burning, aching, gnawing, sore, or sharp. Less common causes include alcohol, cocaine, severe illness and Crohn disease, among others. Colonization of the gastric mucosa with Helicobacter pylori results in the development of chronic gastritis in infected individuals, and in a subset of patients chronic gastritis progresses to complications (e. Gastritis may also occur in those who have had weight loss surgery resulting in the banding or reconstruction of the digestive tract. Evidence does not support a role for specific foods including spicy foods and coffee in the development of peptic ulcers. These drugs used in a short period are not typically dangerous. However, regular use can lead to gastritis. This form of gastritis can occur in more than 5% of hospitalized patients. Brat Diet And GastritisAlso, note that alcohol consumption does not cause chronic gastritis. It does, however, erode the mucosal lining of the stomach; low doses of alcohol stimulate hydrochloric acid secretion. High doses of alcohol do not stimulate secretion of acid. In some disorders the body targets the stomach as if it were a foreign protein or pathogen; it makes antibodies against, severely damages, and may even destroy the stomach or its lining. Gastritis may also be caused by other medical conditions, including HIV/AIDS, Crohn's disease, certain connective tissue disorders, and liver or kidney failure. Since 1. 99. 2, chronic gastritis lesions are classified according to the Sydney system. Gastric ulcers may develop; it is unclear if they are the causes or the consequences. Intestinal metaplasia typically begins in response to chronic mucosal injury in the antrum, and may extend to the body. Gastric mucosa cells change to resemble intestinal mucosa and may even assume absorptive characteristics. Intestinal metaplasia is classified histologically as complete or incomplete. With complete metaplasia, gastric mucosa is completely transformed into small- bowel mucosa, both histologically and functionally, with the ability to absorb nutrients and secrete peptides. In incomplete metaplasia, the epithelium assumes a histologic appearance closer to that of the large intestine and frequently exhibits dysplasia. They include the medications sucralfate and misoprostol. If NSAIDs are being taken regularly, one of these medications to protect the stomach may also be taken. Another cytoprotective agent is bismuth subsalicylate. Several regimens are used to treat H. Most use a combination of two antibiotics and a proton pump inhibitor. Sometimes bismuth is also added to the regimen. See also. The National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK). November 2. 7, 2. Retrieved 1 March 2. Lippincott Williams & Wilkins. ISBN 9. 78. 14. 51. Best Pract Res Clin Gastroenterol. PMID 2. 54. 39. 06. Ferri (2. 01. 2). Ferri's Clinical Advisor 2. Books in 1, Expert Consult - Online and Print,1: Ferri's Clinical Advisor 2. Elsevier Health Sciences. ISBN 9. 78. 03. 23. Lancet (London, England). PMC 5. 38. 89. 03 . PMID 2. 77. 33. 28. Mayo Clinic Gastroenterology and Hepatology Board Review. Oxford University Press. ISBN 9. 78. 01. 99. Adams (2. 01. 2). Emergency Medicine: Clinical Essentials. Elsevier Health Sciences. ISBN 9. 78. 14. 55. Oxford handbook of nutrition and dietetics (2nd ed.). Oxford: Oxford University Press, USA. ISBN 9. 78. 01. 99. PMC 4. 56. 15. 09 . PMID 2. 60. 63. 47. Gastrointestinal endoscopy clinics of North America. PMID 2. 19. 44. 41. Retrieved 2. 00. 8- 1. National Digestive Diseases Information Clearinghouse. National Institute of Diabetes and Digestive and Kidney Diseases. December 2. 00. 4. Retrieved 2. 00. 8- 1. Digestive and Liver Disease. PMID 1. 83. 96. 11. Indigenous microbes and the ecology of human diseases. EMBO Reports. 7 (1. PMC 1. 61. 83. 79 . PMID 1. 70. 16. 44. Strayer, M. D., Ph. D., Professor of Pathology, Department of Pathology and Cell Biology, Jefferson Medical College of Thomas Jefferson University Philadelphia, Pennsylvania ; Founder and Consulting Editor, Emanuel Rubin, M. D., Gonzalo Aponte Distinguished Professor of Pathology, Chairman Emeritus of the Department of Pathology and Cell Biology, Jefferson Medical College of Thomas Jefferson University, Philadelphia, (2. Rubin's pathology : clinicopathologic foundations of medicine (Sixth ed.). Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins. ISBN 9. 78. 16. 05. January 2. 00. 7. Retrieved 2. 00. 9- 0. J Physiol Pharmacol. Suppl 2: 1. 17–3. PMID 1. 88. 12. 63. Jackson Siegelbaum Gastroenterology. Retrieved 2. 00. 8- 1. Gastroenterol J (in German). PMID 2. 67. 96. 57. Retrieved 2. 00. 8- 1. Retrieved 2. 00. 8- 1. Osteopathic Family Physician. PMID 1. 84. 57. 46.
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